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And, of course, timely consultation with a specialist if a disease is suspected. The congenital form of anhidrosis is due to the underdevelopment of the sweat glands. Basically, this species is observed with anhydrotic ectodermal dysplasia. In turn, acquired anhidrosis can be atrophic, asthenic, traumatic, toxic, functional and neurological in nature.
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Diltiazem anhidrosis occurs with beriberi, liver cirrhosis, diabetes mellitus, Addison's disease and others.

Also, the cause of anhidrosis can be various diseases of the nervous system, damage to the spinal cord or peripheral nerves, causing disturbances in the innervation of the sweat glands. Another reason for the development of diltiazem is the use of drugs, for example, anticholinergic or ganglionic blockers, as a result of which the sweating process is disturbed. Acute generalized anhidrosis can be caused by poisoning and intoxication of the body (preuremic conditions, toxicosis of pregnant women, cholera, diarrhea, radiation sickness, food intoxication) and dehydration.

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In acute localized anhidrosis, normally functioning sweat glands can overproduce sweat, so patients complain of excessive sweating rather than dry skin. But this is rather an exception. If for a long time there will be no sweating, atrophy of diltiazem glands occurs. It also increases the risk that the disease will become chronic. Anhidrosis has a number of its own characteristics, so the diagnosis includes special tests with the use of drugs that increase sweating, as well as intradermal administration of histamine.
Collin Manning
Collin Manning
17:16 15 Feb 20

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Acute generalized anhidrosis is characterized by the presence of dry, hot and red skin, fever, rapid heartbeat and respiration, clouding of consciousness, convulsions are often observed.

It is possible to determine the decrease in sweating in a certain part of Diltiazem thanks to special moisture detectors that are installed on the skin. The patient is wrapped in a thermal blanket, after which the detectors begin to record the level of sweating.

In addition, these tests include: axonal reflex test; skin biopsy; the imprint of sweat on the silastic. A disease such as anhidrosis does not always need to be treated. If treatment is necessary, then first of all it is necessary to eliminate the causes that provoke the development of anhidrosis.

Patients are advised to avoid overheating. Dry areas of the skin are lubricated with indifferent ointments and moisturizing creams. To improve the condition of the skin, intramuscular injections of vitamin B12 are used, multivitamins and retinol (vitamin A) are prescribed. Anhidrosis is a pathological condition caused by a malfunction of the sweat glands and manifested by a sharp decrease in sweating up to its complete cessation. The development of congenital anhidrosis is associated with hypoplasia or aplasia of Diltiazem glands. This pathology, in particular, accompanies anhydrotic ectodermal dysplasia.

Anhidrosis often develops against the background of various skin diseases. For example, on the affected areas of the skin with leprosy, ichthyosis, scleroderma and some other diseases, there is a local violation of sweating. Diseases such as deep prickly heat (miliaria profunda) or prickly heat can lead to acute generalized anhidrosis. Often the cause of anhidrosis is damage to the spinal cord, peripheral nerves and certain diseases of the nervous system. In this case, the violation of sweating is due to a violation of the innervation of the sweat glands.

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If the drinking regimen is not followed in the heat, sweating decreases. This condition is regarded as physiological anhidrosis. However, at very high ambient temperatures and insufficient fluid intake, a tropical form of anhidrosis occurs. It can also develop when the ducts of the sweat glands become clogged with dust.

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Against the background of treatment with ganglioblockers or anticholinergics, sweating may stop. This phenomenon is temporary and is considered as a side effect of therapy. Depending on the cause, anhidrosis can be congenital or acquired.

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If the cessation of sweating is observed only in a certain area of ​​​​the skin, this form of the disease is called local anhidrosis. In contrast, in the generalized form, the functions of all sweat glands are disrupted.

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The main symptoms of anhidrosis are increased dryness of the skin and lack of sweating. High dryness of the skin leads to the fact that they crack. Through cracks, pathogens easily penetrate inside, causing infectious and inflammatory diseases.

In accordance with the duration of the course of the disease, chronic and acute anhidrosis are distinguished.

Sweat not only moisturizes the skin, it also removes various toxins from the body. Therefore, the cessation of sweating leads to the development of an intoxication syndrome, which is characterized by an increase in body temperature, nausea and vomiting, as well as vasomotor reactions. At the same time, the body seeks to remove the toxic substances accumulating in it, using the system of other organs.

With local anhidrosis, increased sweating of neighboring skin areas with normally functioning sweat glands is often observed. Some patients with local anhidrosis go to the doctor with complaints not of dry skin, but, on the contrary, of increased sweating.

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With a generalized form of anhidrosis, the functions of all sweat glands are disrupted. Acute generalized anhidrosis is severe. This form of the disease is characterized by: dry, red and hot skin; rapid increase in body temperature; increased heart rate; increased frequency of respiratory movements; disturbances of consciousness; convulsive seizures.

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